الجمعة، 25 يناير 2013


LOCAL COMPLICATIONS OF ANAESTHETICS

A)COMPLICATIONS ARISING FROM DRUGS OR CHEMICAL USED
1.SOFT TISSUE INJURY
2.SLOUGHING OF TISSUES

B)COMPLICATIONS ARISING FROM INJECTION TECHNIQUES
1)NEEDLE BREAKAGE
2)HEMATOMA
3)FAILURE TO OBTAIN LOCAL ANESTHESIA
4)POST-INJECTION HERPETIC LESIONS

C)COMPLICATIONS ARISING FROM BOTH
1)PAIN ON INJECTION
2)BURNING ON INJECTION
3)TRISMUS
4)BLANCHING OF SKIN
5)EDEMA
6)PERSISTENT PARATHESIA OR ANESTHESIA
7)INFECTION
8)PERSISTENT PAIN
9)NEUROLOGICAL SYMPTOMS
               FACIAL N. PARALYSIS
               VISUAL DISTURBANCES


1)SOFT TISSUE INJURY


CAUSES
PREVENTION
MANAGEMENT
-SELF INFLICTED TRAUMA TO LIPS ,TONGUE WHILE STILL NUMB
-SEEN IN CHILDREN AND MENTALLY AND PHYSICALLY DISABLED
-SOFT TISSUE ANESTHESIA LASTS LONGER THEN PULPAL
-APPROPRIATE DURATION LA
-COTTON ROLLS BETWEEN LIPS AND TEETH
-WARN THE PATIENT AND GUARDIAN AGAINST EATING,DRINKING HOT FLUIDS AND BITING ON LIPS OR TONGUE TO TEST FOR ANESTHESIA
-ANALGESICS FOR PAIN
-ANTIBIOTICS
-LUKEWARM SALINE RINSES TO AID IN DECREASE ANY SWELLING THAT MAY BE PRESENT
-PETROLEUM JELLY AS LUBRICANT
2)SLOUGHING OF TISSUES


i)EPITHELIAL DEQUAMATION
-TOPICAL ANESTHETIC FOR PROLONGED PERIOD
-HIGHTENED SENSTIVITY OF TISSUE TO LA REACTION IN AREA OF TOPICAL ANESTHETICS
ii)STERILE ABSCESS
-PROLONGED --ISCHEMIA DUE TO VASOCONSTRICTOR
-DEVELOPS ON HARD PALATE
-DO NOT USE HIGH CONC. LA WITH VASOCONSTRICTOR
(NOREPINEPHRINE 1:30,000 NOT PRESCRIBED)
DEPEND ON INJURY
-SYMPTOMATIC-ANALGESICS,ORABASE
-RESOLVES WITHIN 1-2 WEEKS
-AN ESTABLISH LESION MAY REQUIRE INCISION AND DRAINAGE



B)COMPLICATIONS ARISING FROM INJECTION TECHNIQUES

1)NEEDLE BREAKAGE

CAUSES

PRIMARY CAUSE-
              UNEXPECTED MOVEMENT OF PATIENT
SECONDARY CAUSE-
¤  INAAPROPRIATE THICKNESS OF NEEDLE
¤  PREVIOUSLY BENT
¤  REDIRECTION OF NEEDLES ONCE INSERTED INSIDE TISSUE
¤  MANUFACTURE DEFECT(RARE)
¤  FORCING NEEDLE AGAINST RESISTENCE
¤  NEEDLE ENGAING THE PERIOSTEUM


PREVENTION
v  INFORM THE PATIENT
v  USE PROPER GAUZE NEEDLE(FOR N. BLOCK-25 GAUZE,FOR INFILTRATION-27,25,30 GAUZE
v  USE PRESTERLIZED DISPOSABLE NEEDLES
v  ENTIRE LENGTH SHOULD NOT BE INSERTED(FEW MM AWAY FROM HUB)
v  DO NOT REDIRECT IF EMBEDDED
v  USE GOOD QUALITY NEEDLE
v  GENTLE MANIPULATION-NO EXCESSIVE FORCE
v  DO NOT PERMIT THE NEEDLE TO ENGAGE THE PERIOSTEUM
v  STABILISATION OF JAW
v  NEEDLE SHOULD ALWAYS BE KEPT DURING INSERTION
v  AVOID MULTIPLE PENETRATIONS

MANAGEMENT
CALM,DO NOT PANIC
INFORM PATIENT
IF VISIBLE-USE HEMOSTAT OR MAC GILLS TUBE
IF NOT-FLOUROSCOPE,FOLLOW UP,SURGERY



2)HEMATOMA

THE EFFUSION OF BLOOD INTO EXTRAVASCULAR SPACES CAN RESULT FROM INADVERTENTLY NICKING A BLOOD VESEL(ARTERY OR VEIN)DURING THE INJECTION OF LA

NICKING OF ARTERY-HEMATOMA INCRESE RAPIDLY IN SIZE
NICKING OF VEIN-MAY OR MAY NOT RESULT IN FORMATION

CAUSE
NICK→BLOOD EFFUSES FROM VESSELS UNTIL EXTRAVASCULAR PRESURRE EXCEEDS INTRAVASCULAR→CLOTTING OCCURS

PREVENTION
³  MODIFY INJECTION TECHNIQUE AS DICTATED BY PATIENT
³  USE SHORT NEEDLE(APPROPRIATE LENGTH)
³  MINIMIZE NO. OF PENETRATION
³  NEVER USE NEEDLE AS A PROBE ON TISSUE

MANAGEMENT

IMMEDIATE-DIRECT PRESSURE AT SITE OF BLEEDING FOR NOT LESS THAN 2 MINS
BLOCK
PRESSURE SITE
CLINICAL MANIFESTATION
IANB
MEDIAL ASPECT OF MANDIBULAR RAMUS
INTRAORAL DISCOLORATION AND PROBABLE TISSUE SWELLING ON MEDIAL ASPECT OF MANDIBULAR RAMUS

INFRAORBITAL
INFRAORBITAL FORAMEN
DISCOLORATION OF SKIN BELOW THE LOWER EYELID

MENTAL N. BLOCK
MENTAL FORAMEN
DISCOLORATION OF SKIN OVER THE MENTAL FORAMEN OR SWELLING IN THE MUCOBUCCAL FOLD IN REGION OF MENTAL FORAMEN
PSA N BLOCK
SOFT TISSUE IN MUCOBUCCAL FOLD AS FAR AS POSSIBLE AS DISTALLY AS CAN BE TOLERATED BY PATIENT
COLORLESS SWELLING APPEAR ON SIDE OF FACE (USUALLY A FEW MINUTES AFTER THE INJEVTION IS COMPLETED)→DAYS INFERIOR AND ANTERIOR TOWARD THE LOWER ANT. REGION OF CHEEK

IN PSA HEMATOMA EARLIER IN PTERYGOID VENOUS PLEXUS
ACCORDING TO SICHER –PSA ARTERY

OF SORENESS DEVELOPS –ADVISE THE PATIENT TO TAKE ANALGESIC
DO ON APPLY HEAT OVER FOR AT LEAST 4-6 HOUR(VASODILATION-INCREASES IN SIZE)
HEAT APPLIED ON NEXT DAY-ACTS AS A)ANALGESIC AND B)    VASODILATORè↑RATE AT WHICH BLOOD ELEMENTS ARE RESORBED
IN FORM OF WARM MOIST TOWELS TO THE AFFECTED AREA FOR 20 MIN EVERY HOUR
RESOLVE S WITHIN 7-14 DAYS.


5)FAILURE TO OBTAIN ANESTHESIA

CAUSES
A)OPERATOR DEPENDENT
i)LA AGENT(TYPE,DOSE)
ii)IMPROPER SURGICAL TECH.
iii)INJ OF WRONG SOLN.
iv)I.V
v_I.M

B)PATIENT DEPENDENT
i)ANATOMICAL-ADDITIONAL INNERVATION
ii)PSYCHOLOGICAL –UNCOOPRATIVE,MOVEMENT
iii)PATHOLOGICAL-INFECTION

ADDITIONAL INNERVATION
CUTANEOUS COLLI NERVE’ (CERVICAL CUTANEOUS NERVE)-(A BRANCH OF 3RD CERVICAL NERVE)-ENTERS A SMALL FORAMEN ON LINGUAL ASPECT OD RAMUS AND SUPPLIES INNERVATION TO MANDIBULAR TEETH.
IN CASE OF FAILURE IN OBTAINING OPERATIVE ANESTHESIA AFTER A MANDIBULAR INJ. ,A SUPPLEMENTAL INJ.CAN BE GIVEN TO CERVICAL CUTANEOUS NERVE.THIS IS DONE BY INSERTING THE NEEDLE LINGUALLY BETWEEN 2 BICUSPID TEETH,AT THE REFLECTION OF MUCOUS MEMBRANE AND DIRECTING IT POSTERIORLY,ABOUT HALF OF THE NEEDLE IS INSERTED AND ABOUT 0.5ML OF SOLN. IS INJECTED.

4)POST INJECTION HERPETIC LESIONS

CAUSE
  • REACTIVATION OF DORMANT HERPES VIRUS
  • H/O RECUURENT HERPES LABIALIS
  • IN TERMINAL BRANCHES OF TRIGERMINAL NERVE

PREVENTION-DELAY SURGICAL INTERVENTION IN THE ACTIVE STAGE

MANAGEMENT-ANTIVIRAL DRUGS


C)COMPLICATIONS ARISING FROM BOTH

CAUSES
PREVENTION
MANAGEMENT

1)PAIN ON INSERTION
-CARELESS TECH.
-BLUNT NEEDLE
-RAPID INSERTION OF LA SOLN. CAN CAUSE TISUE DAMAGE
-HIGH TEMP. OF SOLN.
-PROPER TECH.
-SHARP NEEDLE-INSERT LA SLOWLY
-USE STERILE LA SOLN.
-USE TOPICAL LA B4
-SOLN. AT ROOM TEMP.
NOT REQUIRED
2)BURNING SENSATION
-RAPID INJ.
-CONTAMINATED NEEDLE CARTRIDGE
-HIGH TEMP. LA SOLN.
ALTERED PH OF SOLN. (PH PLAIN-5 APP,WITH VASOCONSTRICTOR-3 APP)

-SLOW INJ.
-SOLN. AT ROOM TEMP.
NOT REQUIRED
3)INFECTION
-CONTAMINATION OF NEEDLE
-IMPROPER PREP. OF SITE
-NEEDLE PASSING THROUGH AN AREA OF INFECTION
-LA SOLN DEPOSITED UNDER PRESSURE ,AS IN PDL INJ.→TRANSPORT BACTERIA
-PROPER PREP. OF SITE PRIOR TO PENETRATION
-CAREFUL HANDLING OF NEEDLES (AVOID TOUCHING NON-STERILE SURFACE)
-ANALGESICS
-ANTIBIOTICS
-PHYSIOTHERAPY
-MUSCLE RELAXANTS
4)EDEMA
-TRUAMA
-INFECTION
-ALLERGY
-HEMORRHAGE
-INJ OF IRRITATING SOLN.
-PREOP ASSESMENT
-CAREFUL HANDLING OF LA ARMAMENTARIUM
-ATRAUMATIC TECH.
-FIND OUT CAUSE
-ALLERGY-(A,B,C,D)
5)TISSUE BLANCHING
-TRAUMA TO BLOOD VESSEL BY NEEDLE
-I.V. ADMINISTRATION
-USE ASPIRATION TECH.
-AVOID INTRAARTERIAL ADMINISTRATION
-TRANSIENT PHENOMENON
-NO T/T REQUIRED


6)TRISMUS

CAUSES

PRIMARY CAUSE-TRAUMA TO MUSCLE ,BLOOD VESSELS IN INFRATEMPORAL FOSSA

SECONDARY CAUSES-
#INJECTION OF LA CONTAINING IRRITATING SOLN.(ALCOHOL,COLD STERILISING SOLN.)
#LA HAVE MILD MYOTOXIC PROPERTIES
(AIDS TO PROGRESSIVE NECROSIS OF EXPOSED MUSCLE FIBRES)
# HEMATOMA –(LEADS TO IRITATION OF MUSCLE FIBRES
# LOW GRADE INFECTION
# EXCESSIVE DEPOSITION OF LA-DISTENSION OF TISSUES-POST INJ TRISMUS
#THE BARB OCCURRED WHEN THE NEEDLE COME INTO CONTACT WITH THE MEDIAL ASPECT OF THR MANDIBULAR RAMUS,WITHDRAWL OF THE NEEDLE FROM TISSUE INCREASED THE LIKELIHOOD OF INVOLVEMENT OF THE LINGUAL OR IANB AND DEVELOPMENT OF TRISMUS

PROBLEMS
AVG. INTERINCISAL OPENING IN ACSES OF TRISMUS IS 13.7MM

IN CHRONIC HYPOMOBILTY-
  • IF T/T NOT GIVEN
  • SECONDARY TO ORGANISATION OF HEMATOMA WITH SUBSEQUENT FIBROSIS AND SCAR CONTRACTURE
  • INFECTION –INCRESED PAIN-INCRASED TISSUE REACTION(IRRITATION AND SCARRING).

PREVENTION

Ø  USE SHARP,STERILE,DISPOSABLE NEEDLE
Ø  USE ASEPTIC TECH.
Ø  ATRAUMATIC TECH.
Ø  AVOID MULTIPLE PENETRATION
Ø  USE MINM EFFECTIVE VOL. OF LA


MANAGEMENT

1)HEAT THERAPY-HOT MOIST TOWELS TO AFFECTED AREA FOR 20 MINS EVERY HOUR

2)WARM SALINE RINSE-HELD IN THE MOUTH ON THE INVOLVED SITE AND SPIT OUT

3)ANALGESICS ASPIRIN(325MG)

4)MUSCLE RELAXANTS-
CHLOROXAZONE (250 mg IN 2 TO 3 DIVIDED DOSE)
OR DIAZEPAM (5-10 mg BID)
OR MEMEPROBAMATE(1.2g IN 3-4 DIVIDED DOSES)

5)PHYSIOTHERAPY-OPENING AND CLOSING THE MOUTH ,AS WELL AS LATERAL EXCURSIONS OF THE MANDIBLE FOR 5 MINS EVERY 3 TO 4  HOURS.

6)CHEWING GUMS (SUGARLESS)-TO PROVIDE LATERAL MOVEMENT OF TMJ
7)ANTIBIOTICS

AVOID FURTHER DENTAL T/T IN INVOLVED REGION UNTIL SYMPTOMS RESOLVE AND PATIENT IS COMFORTABLE.

IF DENTAL CARE HAS TO BE CONTINUED –THAN ALTERNATE METHOD OR TECH. FOR ACHIEVING LA MAY BE EMPLOYED.
THE AKINSI MANDIBULAR N. BLOCK PROVIDES RELIEF FORM THA MOLAR DYSFUNCTION AND ALLOWS THE PATIENT TO OPNE THE MOUTH AND PERMITS ADMINSTARTION OF APPROPRIATE ADDITIONAL INJ. IF REQUIRED.
COMPELTE RESOLUTION OD POST INJ. TRISMUS TAKES APPROXIMATELT 6 WEKS ,WITH A RANGE OF 4 TO 20 WEEKS.

7)NEUROLOGICAL SYMPTOMS

A)VISUAL DISTURBANCES
i)SQUINT
ii)DIPLOPIA
iii)TRANSIENT AMAUROSIS
iv)PERAMNENT BLINDNESS

i)DIPOPIA OR DOUBLE VISION
v  LA SOLN. INFILTRATING INTO THE ORBIT TO ANESTHETIC THE EXTRINSIC OCULAR MUSLCES OF THE YES.
v  INTAARTERIAL INJ.-UNCOMMON VASCULAR PATTERNS-(ORBIT IS SUPPLIED EITHER WHOLLY OR PARTLY BY MIDDLE MENINGEAL ARTERY.)
v  NO MANAGEMENT REQUIRED (RESOLVES WITHIN 3 HOURS,OR WHEN EFECT ENDS)

ii)TRANSIENT SQUINT AND DOUBLE VISION
v  PARALYSIS OF EXTRINSIC MUSCLES
LA DIFFUSED INTO ORBIT FROM PTERYPALATINE GANGLION AND INFRATEMPORAL FOSSA VIA INFRAORBTAL FISSURE,EFFECTING OCCULOMOTOR,TROCHLEAR,ABDUCENS NERVE.

v  NO TREATMENT REQUIRED









CAUSES
PREVENTION
MANAGEMENT
FACIAL NERVE PARALYSIS

DIRECTLY LA DEPOSITION IN VICINITY OF 7TH CRANIAL NERVE
1)INFRAORBITAL N. BLOCK
2)PARAPERIOSTEAL OF MAXILLARY CANINE
INDIRECTLY-INTO DEEP LOBE OF PAROTID GLAND IN IANB

FOLLOW STANDARD PROTOCOL
EXPLAIN, REASSURE PATIENT
-UNILATERAL LOSS OF MOTOR FUNCTION-TRANSIENT
-EYE DRESSING GIVEN
-CONTACT LENSES SHOULD BE REMOVED
8)PERSISTENT PARATHESIA OR ANAESTHESIA
-INJECTING CONTAMINATED LA SOLUNTION
-TRAUMA TO N. SHEATH
-HEMORRAHGE AROUND N.
-FOLLOW STANDARD PROTOCAL
-CAREFUL SURGICAL TECH.
-PROPER HANDLING OF CARTRIDGE
-REASSURE THE PATIENT
- VIT B1,B6,B12
-IF DOES NOT RESOLVE THAN REFER FOR SURGERY
9)PERSISTENT PROLONGED PAIN
-POOR SURGICAL TECH.(IN SUPRAPERIOSTEAL TEARING VOL.)
-NEEDLE TIP BARBS
-ISCHEMIC NECROSIS
-MULTIPLE PENETRATIONS
-GOOD SURGICAL TECH.
-AVOID NEEDLE WITH BARBS
-USE VASOCONSTRICTORS WITH MAXIMUN DILUTION
-AVOID MULTIPLE PENETRATION
-SYMPTOMATIC


SYSTEMIC COMPLICATIONS OF LA

CAUSES OF ADVERS EDRUG REACTION

TOXICITY CAUSED BY DIRECT EXTENSION OF THE USUAL PHARMACOLOGICAL EFFECTS OF DRUGS-
1.SIDE EFFECTS
2.OVERDOSE
3.LOCAL TOXIC EFFECTS

TOXICITY CAUSED BY ALTERATION IN RECIPIENT OF THE DRUG
1.A DISEASE PROCESS(HEPATIC DYSFUNCTION,CHF,RENAL DYSFUNCTION)
2.EMOTIONAL DISTURBANCES
3.GENETIC ABBERATIONS(ATYPICAL PLASMA CHOLINESTERASE,MALIGNANT HYPERTHERMIA)

TOXICITY CAUSED BY ALLERGIC RESPONSES TO THE DRUGS

OVERDOSE
A DRUG OVERDOSE REACTIONS HAS BEEN DEFINED AS THOSE CLINICAL SIGNS AND SYMPTOMS THAT RESULT FROM AN OVERLY HIGH BLOOD LEVEL OF A DRUG IN VARIOUS TARGET ORGANS AND TISSUES

PREDISPOSING FACTOR
PATIENT FACTORS,DRUG FACTORS

PATIENT FACTOR
DRUG FACTOR
AGE
WEIGHT
OTHER DRUGS
SEX
PRESENCE OF DISEASE
GENETICS
MENTAL ATTITUDE AND ENVIRONMENT
VASOACTIVITY
CONC.
DOSE
ROUTE OF ADMINISTRATION
RATE OF INJ.
VASCULARITY OF INJ SITE
PRESENCE OF VASOCONSTRICTOR


CLINICAL  MANIFESTAIOTNS OF OVERDOSE

MINIMAL TO MODERATE OVERDOSE LEVELS
SIGNS
SYMPTOMS

q  TALKATIVENESS
q  APPREHENSION
q  EXCITABILITY
q  SLURRRED SPEECH
q  EUPHORIA
q  DYSARTHIA
q  NYSTAGMUS
q  VOMITTING
q  DISORIENTATION
q  LOSS OF RESPONSE TO PAINFUL STIMULI
q  ↑BP
q  ↑HR
q  ↑RR
q  LIGHTHEADENESS AND DIZZINESS
q  RESTLESSNESS
q  NERVOUSNESS
q  NUMBNESS
q  SENSATION
q  METALLIC TASTE
q  VISUAL DISTURBANCES
q  AUDITORY DISTURBANCES
q  LOSS OF CONSCIOUNESS
q  DROWSINESS AND DISORIENTATION



MODERATE TO HIGH OVERDOSE LEVELS
L  SEIZURE
L  CNS DEPRESSION
L  ↓BP
L  ↓HR
L  ↓RR

LIDOCAINE LEVEL
CVS

1.8-5.0 ug/ML
ANTIDYSRRTHMIC ACTIONS
5.0-10.0
MYOCARDIAL DEPRESSION
10.0PLUS
MASSIVE PERIPHERAL VASODILATION,MYOCARDIAL DEPRESSION
CARDIAC ARREST


CNS
0.5-4
ANTICONVULSANT ACTION
4.5-7
CNS DEPRESSION,EXCITATION
7.5-10.0
CNS DEPRESSION ,SEIZURE
10.0 PLUS
GENERALIZED CNS DEPRESSION



CVS EFFECTS

LA (VASODILATOR)
PERIPHERAL RESISTANCE
↓BP(BP=PR*CO)

FURTHER IN LA CONC.

AFFECT N. CONDUCTION OF HEART
MYOCARDIAL CONTRACTILITY
C.O.(CO=HR*SV)

HEART’S NEURONAL CONDUCTION SYS. IS INHIBITED OR COMPLETELY BLOCKED BY LA.
AT TOXIC LEVELS,DEPRESSION OF INTRACARDIAC N. CONDUCTION CAN RESULT IN ATRIOVENTRCULAR DISSOCIATION,VENTRICULAR RHYTHM ,VENTRICULAR FIBRILLATION AND ULTIMATELY CARDIAC ARREST.

CNS EFFECTS

THE CONDUCTION OF INHIBITORY NEURONS ID USUALLY BLOCKED BY LA AGENTS AS THEY REACH TOXIC LEVELS-RESULTING IN UNMODIFIED ACTION OF FACILITATORY NEURONS(IE,CONVULSIVE-LIKE MOVT.)AS THE DOSE INCREASES,FACILITATORY NEURONS ARE ALSO BLOCKED RESULTING IN CESSATION OF FUNCTION.
CERTAIN AMIDE TYPE AGENTS(IE LIDOCAINE)-EFFECT PRIMARILY FACILATORY NEURONS,HENCE DEPRESSION IS SEEN RATHER THAN EXCITATION.


MANAGEMENT

1)MILD OVERDOSE
RETENTION OF CONCIOUSNESS,TALKATIVENESS,AGITATION,
↑HR,↑BP. ↑RR(5-10 MIN)→←

P→A→B→C→D

DEFINITIVE CARE
i)REASSURE THE PATIENT
ii)ADMINISTER OXYGEN VIA NASAL CANULA TO PREVENT ACIDOSIS
iii)MONITOR AND RECORD VITAL SIGNS
iv)ESTABLISH i.v. INFUSION
v)USE OF ANTICONVULSANTS –NOT USUALLY INDICATED
       DIAZEPAM-5mg.MIN i.v.
        MIDAZOLAM-1mg/MIN

2)SEVERE OVERDOSE
UNCONSCIOUSNESS WITH OR W/O CONVULSIONS
RAPID ONSET(WITHIN 1 MINUTE)

i)PROTECT PATIENTS ARMS,LEGS AND HEAD
LOOSEN TIGHT CLOTHES
ii)IMMEDIATELY SUMMON EMERGENCY MEDICAL ASSISTENCE.
iii)CONTINUE BLS
iv)ADMINISTER ANTICONVULSANT
    DIAZEPAM –i.v -5mg/min
IF VENEPUNCTURE NOT FEASIBLE
     MIDAZOLAM-im -1mg
IF HYPOTENSION PERSISTS(30 MINS)-VASOPRESSOR
     (PHENYNEPHRINE OR METHAOXAMINE)IM

EPINEPHRINE OVERDOSE

CLINICAL MANIFESTATIONS
SIGNS-↑BP. ↑HR,CARDIAC DYSRTHYMIAS

SYMPTOMS-FEAR,ANXIETY,THROBBING HEADACHE,PERSPIRATION,WEAKNESS,PALLOR,RESP. DIFFICULTY,PALPITATION

EPINEPHRINE
mg/ml
Mg/CARTRIDGE
MAX NO. OF CARTRIDGES
1:50,000
0.02
0.036
5(H),1(C)
1:100,000
0.01
0.018
10(H),2©
1:200,000
0.005
0.009
20(H),4©


MANAGEMENT
P→A→B→C→D
P-SEMISITIING OR ERECT POSITION( ↓CEREBRAL BP)

i)REAASURE THE PATIENT
ii)MONITOR VITAL SIGNS
iii)OXYGEN ADMINISTERE IF NECESSARY( C/I IN HYPERVENTILATION)
iv)RECOVERY


ALLERGY

ALLERGY IS A HYPERSINSITIVE STATE,ACQUIRED THROUGH EXPOSURE TO A PARTICULAR ALLERGEN,REEXPOSURE TO WHICH PRODUCE HEIGHTENED CAPACITY OT REACTION.

PREDISPOSING FACTORS
z  METHLYPARABEN
z  SODIUM BISULPHITE ALLERGY
z  EPINEHRINE
z  LATEX ALLERGY
z  TOPICAL ANESTHETIC ALLERGY

PREVENTION-PROPER HISTORY

ALLERGY TESTING
0.1ML OF EACH(INTRAVENOUS)
  0.9%NACL

  1% OR 2% LIODCAINE,
  3%MEPIVACAINE
  4%PRILOCAINE(W/O METHYL PARABEN,BISULPHITE,VASOPRESSORS.

INTRAORAL CHALLENGE TEST
0.9 ML OF LA SOLN. SUPRAPERIOSTEAL INFILTRATION ATRAUMATIC(BUT W/O TOPICAL LA)ABOVE A MAXILLARY RIGHT OR LEFT PREMOLAR OR ANT. TOOTH.

DENTAL MANAGEMENT IN CASE OF PRESENCE OF LA ALLERGY:
Í NO T/T OF AN INVASIVE NATURE CARRIED OUT
Í IF EMERGENCY –THEN UNDER GENERAL ANESTHESIA
Í IF GA NOT AVAILABLE –HISTAMINE BLOCKER
DIPPHENHYDRAMINE HCL IN 1 % SOLN. WITH 1:100,000 EPINEPHRINE(30 MIN OF PULPAL ANESTHESIA)
Í NITORUS OXIDE
Í ALTERNATIVES-ELECRONIC DENTAL ANESTHESIA


CLINICAL MANIFESTATIONS OF ALLERGY

DERMATOLOGICAL REACTIONS-
URTICARIA-WHEAL
ANGIOEDEMA-LOCALISED SWELLING INVOLVING FACE,HANDS,FEET,GENITILIA,LIPS,TONGUE.

RESPIRATORY REACTIONS-
BRONNCHOSPASM
RESP. DISTRESS
DYSPNOEA,WHEEZING,FLUSHING,CYANOSIS,PERSPIRATION,TACHYCARDIA,INCREASED ANXIETY,LARYNGEAL EDEMA

GENERALISED ANAPHYLAXIS
SKIN REACTION-PRURITIS,ERYTHEMA,URTICARIA,CONJUCTIVITIS,RHINITIS
GIT DISTURBANCE
RESP STMPTOMS-WHEEZING,DYSPNOEA
CVS-PALLOR,TACHYCARDIA,HYPOTENSION,CARDIAC DYSARRTHYMIA,UNCONCIOUSNESS,CARDIAC ARREST


MANAGEMENT

P→A→B→C→D
i)ADMINISTER EPINEPHRINE 0.3mg IM/SC OR
 HISTAMINE BLOCKER-50mg DIPHENHYDRAMINE OR
10 mg CHLORPHENIRAMINE
ii)MEDICAL CONSULTATION FROM PHYSICIAN
iii)OBSERVE THE PATIENT (60MIN)
iv)PRESCRIBE ORAL HISTAMINE BLOCKER
    50 mg CAP-TDS FOR 3-4 DAYS


BRONCHSPASM
P→A→B→C→D

i)TERMINATE T/T
ii)ADMINISTER OXYGEN (5-6 L/MIN)
iii)ADMINISTER EPINEPHRINE 0.3 mg IM/SC
iv)ADMINISTER HISTAMINE BLOCKER TO MINIMIZE RELAPSE
HISTAMINE BLOCKER-50mg DIPHENHYDRAMINE OR
10 mg CHLORPHENIRAMINE
 v)MEDICAL CONSULTATION

LARYNGEAL ODEMA
P→A→B→C→D
i)ADMINISTER EPINEPHRINE 0.3 mg IM/SC
ii)EMERGENCY MEDICAL SERVICE
iii)MAINTAIN AIRWAY
iv)ADDITIONAL DRUGS
HISTAMINE BLOCKER
-50mg DIPHENHYDRAMINE OR
10 mg CHLORPHENIRAMINE
CORTICOSTEROID-
  100mg HYDROCORTICOSONE IM/IV

GENERALIZED ANAPHYLAXIS
P→A→B→C→D
i)                    EMERGENCY MEDICAL SERVICE
ii)                  EPINEPHRINE (0.3ML OF 1:1000) IM/IV
iii)                OXYGEN AND VITAL SIGNS
iv)                IF DOES NOT IMPROVE SECOND DOSE OF EPINEPHRINE IN 10 MIN
v)               ADDITIONAL DRUGS
                 HISTAMINE BLOCKER
                       -50mg DIPHENHYDRAMINE OR
                          10 mg CHLORPHENIRAMINE
                CORTICOSTEROID-
                        100mg HYDROCORTICOSONE IM/IV
Categories:

0 التعليقات: